Acne : causes and practical management / F. William Danby.

Description based upon print version of record.

4.2.1.1 Mothers' milk

Acne: Causes and practical management; Copyright; Contents; Preface; Practical acne therapy; Genetics; Diet; Hormones; Stress; Comedones (plugs in pores); Blemishes-a brief catalogue; Nodules; Scars and sinuses; Support; Introduction; Nomenclature; The three acnes and grading; Acne vulgaris; Acne rosacea; Acne inversa (hidradenitis suppurativa); Grading the three acnes; Acne vulgaris; Acne rosacea; Acne inversa (hidradenitis suppurativa); References; Chapter 1 The three acnes and their impact; 1.1 Acne vulgaris; 1.1.1 Terminology; 1.1.2 The starting point; 1.2 Acne rosacea

1.2.1 The'pimply' part1.2.2 The'redness' part; 1.2.3 The third part, the firm fibrosis; 1.2.4 Part four-ocular rosacea; 1.2.5 Putting it all together; 1.2.6 The inflammatory epiphenomena in acne rosacea; 1.2.7 The'acne rosacea' versus'rosacea' controversy; 1.2.8 Summary; 1.3 Acne inversa (formerly hidradenitis suppurativa); 1.3.1 Before the rupture, where and why?; 1.3.2 After the rupture, what next?; 1.3.3 So what invaders are important in acne inversa?; 1.3.4 What makes this disease behave so much worse than acne vulgaris?

1.3.5 So what can one possibly do to settle down all this inflammation?1.3.6 So how do you get rid of all this material?; 1.3.7 What does the future offer?; 1.4 The psychology of acne; 1.4.1 Acne as a stress; 1.4.2 Acne and self-image; 1.4.3 Isotretinoin therapy and the psyche; 1.4.4 The isotretinoin-depression question; 1.4.5 Isotretinoin in perspective; References; Chapter 2 The folliculopilosebaceous unit-the normal FPSU; 2.1 Anatomy; 2.2 Genetics; 2.2.1 Acne vulgaris; 2.2.2 Acne rosacea; 2.2.3 Acne inversa/hidradenitis suppurativa (AI/HS); 2.2.4 The scottish twins; 2.3 Epigenetics

2.3.1 The farmer's boys2.4 Embryology; 2.5 Histology; 2.5.1 Onwards and downwards; 2.5.2 What is going on inside the FPSU?; 2.6 Physiology; 2.6.1 Hair first; 2.6.2 Oil second; 2.6.3 Last but definitely not least: the follicle; 2.6.4 Looking deeper; 2.7 Biochemistry; 2.8 Hormones, enzymes, receptors, and the intracrine system; Patient Tip Box; 2.8.1 The intracrine system; 2.9 FoxO1 and mTORC1; 2.9.1 The next step; 2.9.2 The broad view; References; Chapter 3 Pathogenetic mechanisms summarized; 3.1 Acne vulgaris; 3.2 Acne rosacea; 3.3 Acne inversa/hidradenitis suppurativa (AI/HS)

3.4 Other variants3.4.1 Malassezia folliculitis; 3.4.2 Eosinophilic pustular folliculitis (ofuji's disease); 3.4.3 Dissecting terminal folliculitis; 3.4.4 Acne keloidalis; 3.4.5 Epidermal growth factor receptor (EGFR) inhibitor eruption; 3.4.6 Acné excoriée des jeunes filles; References; Chapter 4 The acne hormones; 4.1 The endogenous hormones; 4.1.1 Androgens and their sources; 4.1.2 Estrogens and their sources; 4.1.3 Progesterone and the progesteroids; 4.1.4 Insulin; 4.1.5 Growth hormone and insulin-like growth factor-1; 4.2 The exogenous hormones; 4.2.1 Anabolic steroids

Learn to accurately diagnose, prevent and treat all three acnes using both traditional and novel approaches to understanding the causes and selecting the most effective treatments. Acne vulgaris is an extremely common condition. It is troublesome to manage, often persisting into middle age. Exact causes are becoming clear and include several hormonal stimulants, some triggered by the Western diet, and a pathogen ignored for decades. Acnes rosacea and inversa (hidradenitis suppurativa) are discussed from entirely new viewpoints. Acne: Causes and Practical Management will provide readers a.